Dementia type tauopathies, including Alzheimer’s disease are accompanied by an accumulation of abnormal form of tau protein in the brain. By interacting with the filaments called microtubules, this protein plays an important role in non-pathological conditions as it helps the neurons to function correctly. The French team of Etienne-Emile Baulieu recently demonstrated that another protein may be involved in cellular degeneration related to dementias. This protein, commonly named FKBP52, interacts with the tau protein and it seems that its quantity is inversely correlated with tau’s. Simply said, the lower the protein FKBP52 is present, the higher the tau protein is present. This discovery offers crucial elements to understand this kind of disease as well as new clues for detecting and above all healing the disease.
Source: postnoon.com